Vitamin B12 Can Possibly Protect Brain Cells from Getting Damaged by Alzheimer’s


Alzheimer’s is a common term and disease that only does one thing, that is, neutralize brain cells. It is a type of progressive neurological disorder that impacts the memory of a person and impacts brain functioning too. It is a form of a disease known as dementia. Dementia is a type of disease in which a person goes through a state of diminishing memories. Also, starts lacking in behavioral and social skills.

Current reports suggest that around 44 million people are suffering from Alzheimer’s disease or another form of dementia worldwide. According to the National Institute of Aging, Alzheimer’s doubles every five years cycle. And, majorly happens to those who are beyond 65-years of age.

Vitamin B12


For those who don’t know, Alzheimer’s is a non -preventable disease. Scientists still couldn’t find its actual cause, but they suggest that poor lifestyle habits, sex, age, genes, or an injury to the brain play an important role in triggering the condition. Meaning, it is impossible for a human to predict the cause and onset of it.

There are certain ways to modify its causes. Scientists suggest that exercising regularly, consuming freshly prepared food, keeping oneself safe from chronic diseases can help in modifying the risk factors associated with it.

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What Does the Study Show? Could Vitamin B12 Protect Against the Disease?

As of now, scientists couldn’t find a complete treatment for Alzheimer’s, but they are spending lots of hours in labs to find a cure for this disease. A recent study by a team of researchers of the University of Delaware was successful in finding that Vitamin B12 could lessen the impact of Alzheimer’s and will protect brain cells from getting damaged.

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According to science, patients going through this condition are tend to have a protein in their brain, called amyloid-beta. This protein is responsible for imposing a negative impact on neurons and breaking cell-to-cell communication.

Vitamin B12

To study deeply about this matter, researchers used C. elegans. These are tiny soil-dwelling worms and once they start maturing within 36 hours, a gathering of amyloid-beta takes place in them which further causes paralysis. And, when researchers fed with E. coli bacteria, they witnessed opposite results. E. coli bacteria contains high levels of vitamin B12, so it helped worms by protecting against paralysis.

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The lead researcher of this study, Professor Jessica Tanis commented that the worms are either moving or they are not. Meaning, when the worms were given vitamin B12 that was vitamin B12 deficient, paralysis did occur but at a slow pace. Hence, she concluded that this trick worked only if the worms were vitamin B12 deficient in the first place.



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